Austin V. Stone, MD, PhD

Austin Stone, MD photo

Austin V. Stone, M.D., Ph.D.

 E-mail address:ausstone@wakehealth.edu

Education:

PhD, August of 2013, Molecular Medicine and Translational Sciences, Wake Forest School of Medicine, Winston Salem, NC
MD, 2010, University of Cincinnati College of Medicine, Cincinnati, OH
BS, 2005, Wake Forest University, Winston-Salem, NC

Research Interests:

Molecular aberrations in response to joint injury, role of meniscus in the development of osteoarthritis, microtrauma and adaptations in the pediatric skeleton, botulinum neurotoxin in vascular and musculoskeletal disorders, and development of an osteoinductive bone scaffold.

Awards:

Won Resident Basic Science Gold Award, February 2013, for abstract "Inflammatory Factors Associated with Injury Stimulat Early Osteoarthritis Pathways in the Meniscus".

First Prize – Orthopaedic Research and Education Foundation/Orthopaedic Research Society Mid-Atlantic Resident Research Symposium hosted by the Department of Orthopaedic Surgery, Wake Forest School of Medicine:  A potential mechanism for meniscus involvement in osteoarthritis. October 26, 2011

Runner-up, Translational Science Award, WFU Graduate School of Arts and Sciences March 2011

American Orthopaedic Society for Sports Medicine Young Investigator Travel Award September 2010

Grants:

American Orthopaedic Society for Sports Medicine March 2011
Young Investigator Grant, Principle Investigator
Molecular Mechanisms in Meniscus Injury that Contribute to Subsequent Osteoarthrits.

Orthopaedic Research & Education Foundation (OREF) February 2011
Resident Clinician Scientist Training Grant, Principle Investigator
The Role of HIF-2α and NF-κB in meniscus degeneration.

Abstracts:

Stone AV, Stringer KF, Glos DL, Sheppard ZR, Little KJ, Wall, EJ. Repetitive Physiologic Stresses Create Osteochondral Lesions in Juvenile Rabbits. ORS 2011

Stone AV, Glos DL, Stringer KF, Sheppard ZR, Little KJ, Wall EJ. Repetitive Stresses Generate Juvenile Osteochondritis Dissecans-Like Changes in Immature Animals. AAOS 2011.

Stone AV, Glos DL, Stringer KF, Sheppard ZR, Little KJ, Wall EJ. Repetitive physiologic stresses generate osteochondral lesions. POSNA 2010.

Stone AV, Long DL, Loeser RF, Ferguson CM, Human osteoarthritic and degenerative monkey menisci have differential increases of  matrix metalloproteases compared to both osteoarthritic chondrocytes and healthy podium monkey menisci. EOA 2011

Stone AV, Long DL, Loeser RF, Ferguson CM, A potential mechanism for meniscus involvement in degenerative joint disease. NCOA 2011. 

Stone AV, Long DL, Loeser RF, Ferguson CM, A potential biological mechanism for meniscus involvement in osteoarthritis. COS 2011 

Presentations:

Repetitive Stresses Generate Juvenile Osteochondritis Dissecans-Like Changes in Immature Animals. Annual meeting of the American Academy of Orthopaedic Surgeons, San Diego, CA. February 2011 

Differential matrix metalloprotease expression in human osteoarthritic meniscus cells versus osteoarthritic chondrocytes stimulated with inflammatory cytokines Wake Forest Institute for Regenerative Medicine. March 2011 

Quick Reference

Contact Information
Molecular Medicine and Translational Science Graduate Program

Office 336-713-4259

Kay Collare

336-713-4259

kcollare@wakehealth.edu

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